Vascular: Aortic Dissection Aortic Dissection BasicsIntimal Disruption Allows Blood Extravasation Between the Layers of the WallOriginationAscending Aorta (50-65% – Most Common)Descending Aorta (20-30%)Aortic Arch (< 10%)High MortalityAscending Dissection – From Cardiac Failure (Tamponade, Regurgitation/Insufficiency or Coronary Occlusion)Most Common Cause of Death: Cardiac FailureDescending Dissection – From End Organ Failure by Vessel ObstructionPathophysiologyInitial Lesion: Intimal TearBlood Extravasation Between the Layers of the Vessel WallTypically Spreads Antegrade but Can Spread Retrograde“False Lumen” Created Between “Intimal Flap” & Remaining WallMay Have Distal Fenestrations (Intimal Connections) to Maintain PatencyMalperfusion SyndromeOccurs When Branch Occlusion Causes End-Organ IschemiaDynamic ObstructionMore Common (80%)Etiologies:Insufficient Flow Through the True Lumen – Varies with Vessel Circumference, Blood Pressure, Heart Rate and Peripheral ResistanceMobile Intimal Flap Prolapse Occluding Branch Vessel OstiumObstruction is Intermittent in Nature & Responds Better to Beta-Blocker ManagementStatic ObstructionEtiology: Narrowing/Occlusion of Branch Vessels from False Lumen Protrusion into the Branch Vessel with Associated ThrombosisObstruction is Consistent Aortic Dissection Specimen and Intravascular Imaging 1 ClassificationDeBakey Classification Mn Type I: Both – Tear Originates in Ascending & Extends to DescendingType II: Ascending – Tear Originates in Ascending & Confined to AscendingType III: Descending – Tear Originates in DescendingIIIa: Confined to Thoracic AortaIIIb: Originates in Thoracic Aorta & Extends to the Abdominal AortaStanford Classification Mn Type A Aortic Dissection (TAAD): Originates in the Ascending AortaIncludes DeBakey Type I & IIType B Aortic Dissection (TBAD): Originates in the Descending AortaIncludes DeBakey Type III DeBakey Classification 1 Stanford Classification 1 Risk FactorsHypertension (70%)Male Sex (4:1)Aortic Wall Abnormality (Bicuspid Aortic Valve, Coarctation)Cystic Medial NecrosisConnective Tissue Disorders (Marfan & Ehlers-Danlos)PregnancyCocaineTraumaPresentationPain (Back/Chest/Abdomen) – Most CommonPulse DeficitLimb IschemiaMesenteric IschemiaCardiac TamponadeAortic InsufficiencyMyocardial InfarctionSyncope/StrokeDiagnosisGold Standard: CTAHighest Sensitivity: TEE Aortic Dissection on CTA 1 Aortic Dissection – Treatment Initial ManagementInitial Tx: β-Blockers & Vasodilator (Sodium Nitroprusside)Reduce Systolic Blood Pressure & Pulsatile Load/Aortic Stress (dP/dt – Derivative of Pressure/Time from Left Ventricle)Give β-Blockers Before Vasodilator – Avoid Reflex Sympathetic StimulationAgent of Choice: Esmolol for Goal Systolic Blood Pressure < 120 mmHg within 20 MinutesType ARepair All Type A Aortic Dissections Mn Access: Median SternotomyMay Need Cervical/Supraclavicular Incisions to Visualize Great VesselsProcedure: Excision with Interposition Synthetic/Dacron GraftMay Also Require Aortic Valve Replacement/RepairType BUncomplicated: Medical Management*Role of Endovascular Stenting in Uncomplicated Patient’s is EvolvingComplicated: Thoracic Endovascular Aortic Repair (TEVAR)Indications:Malperfusion SyndromeRefractory Severe HypertensionRefractory Severe Chest PainProgression/ExpansionImpending RuptureRuptured: Open Surgical RepairAccess: Left Posterolateral ThoracotomyMay Need Thoracoabdominal IncisionPostoperative ComplicationsAortic AneurysmRisk: 30%ParaplegiaFrom Intercostal Artery OcclusionRecurrent DissectionMost Common in Marfan Syndrome“Bird’s Beaking” of EndograftProximal Edge Pulled Away from Aortic WallAllows for Recurrent DissectionRisks Graft Migration, Collapse or Occlusion Mnemonics DeBakey ClassificationDissections are B.A.D.1 – Both2 – Ascending3 – DescendingStanford ClassificationA-A: Type A – AscendingRepair of Aortic DissectionsA-A: All Ascending Are Repaired References Tran TP, Khoynezhad A. Current management of type B aortic dissection. Vasc Health Risk Manag. 2009;5(1):53-63. (License: CC BY-NC-3.0)