Fluids, Electrolytes & Nutrition: Calcium (Ca)
General Calcium Physiology
Vitamin D
- Cholecalciferol (Vitamin D3) Source
- Ultraviolet (UV) Light Converts 7-Dehydrocholesterol to Pre-Vitamin D3
- By Photolysis
- Pre-Vitamin D3 Spontaneously Isomerizes to Cholecalciferol (Vitamin D3)
- Cholecalciferol Can Also Be Ingested
- Ultraviolet (UV) Light Converts 7-Dehydrocholesterol to Pre-Vitamin D3
- Vitamin D Activation
- Liver Converts Cholecalciferol (Vitamin D3) to 25-Hydroxycholecalciferol (Calcifediol)
- By Hydroxylation
- Kidney Converts 25-Hydroxycholecalciferol (Calcifediol) to 1,25-Hydroxycholecalciferol (Calcitriol)
- By Hydroxylation
- Liver Converts Cholecalciferol (Vitamin D3) to 25-Hydroxycholecalciferol (Calcifediol)
- Calcitriol (1,25-Hydroxycholecalciferol)
- The Active Form of Vitamin D
- Increases Calcium & Phosphate
- Increases Intestinal Calcium & Phosphate Absorption (Primary Method)
- Increases Renal Reabsorption of Calcium
- Stimulates Calcium Release from Bone (Acts on Osteoblasts to Release RANKL which Activates Osteoclasts)
Parathyroid Hormone (PTH)
- *See Endocrine: Parathyroid Anatomy & Physiology
- Increases Calcium & Decreases Phosphate
Calcitonin
- *See Endocrine: Thyroid Anatomy & Physiology
- Decreases Calcium & Phosphate
Hypercalcemia
Causes
- Most Common Causes (90%):
- Hyperparathyroidism – Most Common Benign/Outpatient Cause
- Malignancy
- Breast Cancer – Most Common Malignant Cause
- Squamous Cell Carcinoma of the Lung
- Lytic Bone Lesions
- Less Common Causes:
- Vitamin D Intoxication
- Thiazide Diuretics
- Lithium
- Familial Hypercalcemic Hypocalciuria (FHH)
- Milk-Alkali Syndrome
Presentation Mn
- Stones:
- Kidney Stones
- Gallstones
- Bones:
- Osteoporosis
- Bone Pain
- Groans:
- Abdominal Pain
- Peptic Ulcer Disease (PUD)
- Pancreatitis
- Constipation
- Anorexia
- Nausea & Vomiting
- Thrones:
- Polyuria (Urinary Frequency)
- Polydipsia
- Psychiatric Overtones:
- Fatigue
- Delirium
- Coma
- EKG Changes:
- Shortened QT Interval
- Bradycardia
Severity
- Mild ≥ 10.5 mg/dL
- Moderate ≥ 12.0 mg/dL
- Severe/Crisis ≥ 14.0 mg/dL
Treatment
- Mild: Nothing
- Moderate-Severe: IV Normal Saline & Furosemide
- Also Consider:
- Bisphosphonates
- Calcitonin
- Dialysis
- Also Consider:
Related Pathology
- Familial Hypercalcemic Hypocalciuria (FHH)
- Cause: CaSR (Calcium-Sensing Receptor) Inactivating Mutations
- Autosomal Dominant Inheritance
- Effect: Parathyroid Gland Less Sensitive to Calcium
- Higher than Normal Calcium Levels are Required to Inhibit PTH Release
- Labs:
- PTH Normal-Elevated
- Calcium High
- Urine Calcium Low (Ca/Cr Clearance Ratio < 0.01)
- Ca/Cr Clearance Ratio = (24-Hour Urine Ca/Serum Ca) / 24-Hour Urine Cr/Serum Cr)
- Treatment: Treat Hypercalcemia, No Surgical Treatment Required
- Cause: CaSR (Calcium-Sensing Receptor) Inactivating Mutations
- Milk-Alkali Syndrome
- Cause: Excessive Ingestion of Calcium with Absorbable Alkali (Vitamins/Milk)
- Most Common in Older Women Taking Calcium Carbonate to Treat Osteoporosis
- Most Often Asymptomatic
- Labs:
- High Calcium
- Alkalosis
- Acute Kidney Injury (AKI)
- Diagnosis Based on History
- Treatment: Stop Offending Agent & Treat Hypercalcemia
- Malignancy
- PTH-Related Protein (PTHrP)
- Released in Malignancy with Rapid Onset Hyperparathyroidism
- The Most Common (80%) Cause of Hypercalcemia in Malignancy
- Most Common Causes:
- Breast Cancer
- Squamous Cell Carcinoma (Lung, Head or Neck)
- Ovarian Cancer
- Lytic Bone Lesions
- Increases Calcium from Bone Breakdown
- Less Common (80%) Cause of Hypercalcemia in Malignancy
- Most Common Causes:
- Multiple Myeloma
- Breast Cancer Metastases
- Other Causes of Hypercalcemia in Malignancy:
- Ectopic PTH Secretion
- Increased 1,25-Hydroxycholecalciferol (Calcitriol) Production – Lymphoma
- PTH-Related Protein (PTHrP)
Hypocalcemia
Causes
- Parathyroid Injury During Thyroidectomy – Most Common Cause
- Parathyroidectomy
- Autoimmune Parathyroid Destruction
- Blood Transfusion (Chelation)
- Hypomagnesemia
- Vitamin D Deficiency
- Chronic Kidney Disease (CKD) Induced Vitamin D Deficiency
- Medications:
- Lactate
- Foscarnet
- Bisphosphonates
- Denosumab
- Cinacalcet
- Chemotherapy
- Acute Pancreatitis
- Surgery
- Sepsis or Severe Illness
Presentation
- Perioral Tingling/Numbness – First Sign
- Tetany
- Chvostek Sign (Cheek Tap > Spasm)
- Trousseau Sign (BP Cuff Inflation > Carpopedal Spasm)
- Seizures
- Anxiety & Depression
- Prolonged QT Interval
Treatment
- Primary Treatment: Calcium Replacement
- Correct Any Underlying Hypomagnesemia as Well
Pseudohypocalcemia
- Definition: Falsely Low Calcium from Low Protein Levels (Particularly Albumin)
- Total Calcium Low from Decreased Protein-Bound Calcium
- Ionized Calcium Remains Stable
- Corrected Calcium = Calcium + [0.8 x (4 – Albumin)]
- Normal Ionized Calcium: 4.5-5.5 mg/dL
Mnemonic
Symptoms of Hypercalcemia
- “Stones, Bones, Groans, Thrones & Psychiatric Overtones”