Increased Bilirubin Production – High Indirect Component
- Breast Mild Jaundice
- Benign Hyperbilirubinemia Persisting Beyond 2-3 Weeks
- When Infant Only Given Human Breast Milk
- Mechanism Uncertain
- Lactation-Failure Jaundice
- Cause: Lactation Failure Causing Hypovolemia & Weight Loss
Conjugation Deficiency – High Indirect Component
- Benign Neonatal Hyperbilirubinemia (Physiologic Jaundice of Newborn)
- Cause: Immature UDP-Glucuronosyltransferase
- Normal Transition Seen in Newborn
- Takes 14 Weeks to Reach Adult Levels
- Tx: Phototherapy vs None
- Gilbert’s Syndrome
- Cause: Mild UDP-Glucuronosyltransferase Defect
- Most Common Inherited Bilirubin Metabolism Disorder
- Tx: None
- Crigler-Najjar Syndrome
- Cause: Severe UDP-Glucuronosyltransferase Defect
- Type I – No Conjugation
- Type II – Very Low Activity
- Tx: Lifelong Phototherapy; Liver Tx Curative
Excretion Deficiency – High Direct Component (Other LFT’s Normal)
- Rotor’s Syndrome
- Cause: Hepatic Storage Defect – Conjugated Bilirubin Diffuses into Plasma
- Dx: High Urinary Coproporphyrin, < 70% Isomer I
- Tx: None
- Dubin-Johnson Syndrome
- Cause: Hepatocyte Secretion Defect
- Liver is Grossly Pigmented/Black
- Dx: Normal Urinary Coproporphyrin, > 80% Isomer I
- Tx: None