Oncology: Colorectal Cancer & Polyposis Syndromes
Colorectal Cancer & Polyposis Syndromes
Syndromes
- Familial Adenomatous Polyposis (FAP)
- Lynch Syndrome (Hereditary Nonpolyposis Colon Cancer/HNPCC)
- Juvenile Polyposis Syndrome (JPS/Familial Juvenile Polyposis)
- MUT Y Homolog (MUTYH)-Associated Polyposis (MAP)
- Peutz-Jeghers Syndrome
- Serrated Polyposis Syndrome (SPS)/Hyperplastic Polyposis Syndrome (HPS)
- PTEN Hamartoma Tumor Syndromes:
- Cowden Syndrome
- Bannayan-Riley-Ruvalcaba Syndrome
- Cronkhite-Canada Syndrome
Familial Adenomatous Polyposis (FAP)
Basics
- Autosomal Dominant
- Mutation: APC Gene
Presentation
- 100’s-1,000’s of Colorectal Polyps
- Also See Duodenum Polyps with Predilection of the Ampulla/Periampullary Region
- Colorectal Cancer Risk: Nearly 100% By Age 40
- The Adenomas Themselves are More Abundant but Do Not Have an Individually Higher Risk of Malignancy
- Timing/Progression:
- Onset of Polyps: Puberty
- Diffuse Polyposis: 20-30’s
- Colorectal Cancer Diagnosis: Average Age 39
- Extracolonic Manifestations:
- Desmoid Tumors
- Osteomas
- Sebaceous Cysts
Variants
- Gardner’s Syndrome: FAP with Extracolonic Manifestations
- *Originally Described Colonic Polyposis with Extracolonic Manifestations but Now FAP & Gardner Syndrome are Essentially Interchangeable
- Turcot’s Syndrome: FAP with Brain Tumors
- Medulloblastoma & Glioma Most Common
- Attenuated FAP: Oligopolyposis (10-99 Adenomas) with Later Onset (40-50’s)
- Lower Risk of Colorectal Cancer (80%)
- Gastric Adenocarcinoma & Proximal Polyposis of the Stomach (GAPPS): 100’s of Stomach Polyps
- Polyps Develop in the Body & Fundus but Spare the Antrum
- Generally Do Not See Colorectal Polyps
Diagnosis
- Clinical Diagnosis Often Clear on Colonoscopy
- Flexible Sigmoidoscopy Alone is Often Sufficient
- Primary Diagnosis: APC Gene Mutation
Screening
- Flexible Sigmoidoscopy Every Year, Starting at Age 10-12
- *Continues Even After Colectomy
- Screening EGD Every 2-3 Years After Age 25
- *Or Start Whenever Colon Polyps are Fist Seen (Whichever Comes First)
Surgical Management
- Colectomy Indications (2015 ACG Guidelines):
- Documented or Suspected Colorectal Cancer
- Significant Symptoms
- Adenomas:
- Adenomas with High-Grade Dysplasia
- Significant Increase in Number of Polyps on Consecutive Exams
- Multiple Large Adenomas > 6 mm
- Inability to Adequately Survey the Colon Because of Multiple Diminutive Polyps
- Consider Prophylactic Colectomy by Age 20-25 if Otherwise Not Indicated
- Surgery Options:
- Total Abdominal Colectomy with Ileorectal Anastomosis (IRA)
- Spares Rectum – 5-25% Risk of Future Rectal Cancer
- Only Considered for Mild Polyposis Burden
- Requires Flexible Proctoscopy Every 6-12 Months
- Total Proctocolectomy with Ileal Pouch Anal Anastomosis (IPAA)
- Resects Majority of the Rectum
- Requires Flexible Pouchoscopy Every 6-12 Months
- Total Proctocolectomy with End Ileostomy
- Requires Flexible Endoscopy of Ostomy Every Year
- Total Abdominal Colectomy with Ileorectal Anastomosis (IRA)
FAP on Colonoscopy 1
Lynch Syndrome (Hereditary Nonpolyposis Colon Cancer/HNPCC)
Basics
- Autosomal Dominant
- Mutations: MLH1, MLH2, MSH6 or PMS2
- DNA Mismatch Repair Genes
- Cause Microsatellite Instability (Genetic Hypermutability)
- 20% are Sporadic Mutations
Types
- Type I: Colorectal Cancer
- Type II: Colorectal Cancer & Extracolonic Malignancy
Presentation
- Colorectal Cancer
- Most Common Site: Right Colon (Sporadic Cancers are Most Common on the Left)
- Lifetime Risk: 12-90% Depending Mutation & Associated Risk Factors
- Endometrial Cancer
- Most Common Extracolonic Malignancy
- Other Less Common Extracolonic Malignancies:
- Ovarian Cancer
- Stomach Cancer
- Small Bowel Cancers
- Pancreatic Cancer
- Kidney Cancer
- Bladder Cancer
- Brain Tumors
Amsterdam Criteria
- Family History-Based Criteria to Help Identify Patients at Risk for Lynch Syndrome
- “3-2-1 Rule”
- 3 Relatives Affected
- 2 Generations Affected
- 1 Before Age 50
- Low Sensitivity
- *Amsterdam Criteria is Not Diagnostic, Diagnosis Confirmed by Genetic Screening
Screening
- Colonoscopy Every 1-2 Years, Starting at Age 20-25
- EGD Every 2-4 Years, Starting at Age 30-35
- Pelvic Exam & Endometrial Biopsy Every Year, Starting at Age 30-35
Surgical Management
- Surgical Options for Colorectal Cancer or Unresectable Adenomas:
- Total Abdominal Colectomy with Ileorectal Anastomosis (IRA)
- Requires Flexible Proctoscopy Every Year
- Total Proctocolectomy with Ileal Pouch Anal Anastomosis (IPAA)
- Total Proctocolectomy with End Ileostomy
- Total Abdominal Colectomy with Ileorectal Anastomosis (IRA)
- Risk-Reducing TAH-BSO is Indicated for Females After Age 40 or at Completion of Childbearing
Other Syndromes
Juvenile Polyposis Syndrome (JPS/Familial Juvenile Polyposis)
- Mutations: SMAD4 (MADH4) or BMPR1A
Mn- Autosomal Dominant
- Presents with Multiple Hamartomas Throughout the GI Tract
- Most Patients Have Symptoms by Age 20 (Anemia/GI Bleeding)
- Risk or Colorectal Cancer: 10-40%
- Also at Increased Risk for Stomach & Small Intestinal Cancers
- Surveillance: Colonoscopy Every 1-3 Years, Starting at Age 12
- Also Consider Concurrent EGD Surveillance
MUT Y Homolog (MUTYH)-Associated Polyposis (MAP)
- Mutation: MYHH Gene
- Autosomal Recessive
- Characterized by 10-100 Colorectal Adenomatous Polyps
- Often Right-Side Predominant
- Risk or Colorectal Cancer: 60-75%
- Extracolonic Manifestations:
- Gastric Polyps
- Duodenal Polyps
- Desmoid Tumors
- Osteomas
- Thyroid Cancer
- Retinal Pigment Epithelial Hypertrophy
- Epidermal Cysts
- Surveillance: Colonoscopy Every 1-2 Years, Starting at Age 20-30
Serrated Polyposis Syndrome (SPS)/Hyperplastic Polyposis Syndrome (HPS)
- Definitions (WHO Criteria):
- > 20 Serrated Polyps
- Any Serrated Polyps Proximal to the Sigmoid Colon with a Family Hx of SPS
- ≥ 5 Serrated Polyps > 5 mm Proximal to the Rectum with ≥ 2 > 10 mm
- Mutations: RNF43 (Most Closely Related), SMAD4, BMPR1A, PTEN or MUTYH
- Most Patients Have No Predisposing Mutation
- Risk or Colorectal Cancer: 25-40%
Peutz-Jeghers Syndrome
- *See Oncology: Hereditary Cancer Syndromes
- Risk or Colorectal Cancer: 40%
PTEN Hamartoma Tumor Syndrome
- Includes:
- Cowden Syndrome
- Bannayan-Riley-Ruvalcaba Syndrome
- Cronkhite-Canada Syndrome
- *See Oncology: Hereditary Cancer Syndromes
- Risk or Colorectal Cancer: 10-18%
Comparisons
Colorectal Cancer & Polyposis Syndromes
Colorectal | Stomach/SI | Osteoma | Desmoid | Thyroid | Endometrium | Breast | Other | |
FAP | X | X | ||||||
Turcot | X | X | Brain Tumors | |||||
Gardner | X | X | X | X | Sebaceous Cysts | |||
Lynch | X | X | X | |||||
MAP | X | X | X | X | X | Eye & Epidermoid Cyst | ||
Cowden | X | X | X | X | X | Kidney | ||
Peutz-Jeghers | X | X | X | X | Melanin Spots, Pancreas & Testicles | |||
JPS | X | X | ||||||
SPS | X |
- Not Included: Bannayan-Riley-Ruvalcaba Syndrome & Cronkhite-Canada Syndrome
Mnemonics
Familial Juvenile Polyposis
- Juveniles Get “MAD” (Mutation: SMAD4/MADH4)
References
- Makarewicz W, Bobowicz M, Sawicka W, Rzyman W. The treatment of chronic pleural empyema with laparoscopic omentoplasty. Initial Report. Wideochir Inne Tech Maloinwazyjne. 2014 Dec;9(4):548-53. (License: CC BY-NC-ND-3.0)